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Biological validation demonstrated superior therapeutic outcomes compared to monotherapy or free drug combinations, including enhanced apoptosis induction, elevated reactive oxygen species generation, cell cycle arrest in breast cancer cells, and significant tumor regression with metastasis suppression in murine models without systemic organ toxicity
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Therefore, to safely harness the attractive catabolic and thermogenic effects of glucagon for treating metabolic disease, a counter-balancing therapy that selectively opposes the risk for glucagon-induced hyperglycaemia is required
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