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By restoring cellular methyl donor capacity, it may support genomic stability and longevity-associated gene expression patterns
A.CaiY.KerckhofsK.LingZ.MartensG.HeimbergH.et al (2004)
Consistent with an iron-sulfur cluster defect, SLC25A39/40 knock out cells displayed reduced activity of the enzyme aconitase, in which iron sulfur clusters function in both substrate binding and catalysis (Fig 4f)
(2014) reported a 36% increase in keratin gene expression, contributing to stronger hair shafts
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