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Furthermore, Lca inhibited LPS-elevated expression of pro-inflammatory cytokines IL-6, TNF-, IL-17A, and IL-1 in macrophages and IL-6 and KC in the BALF of ALI mice, which were all related to the formation and development of ALI/ARDS ( 25 , 26 )
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Additionally, rejection-associated macrophages demonstrated altered amino acid metabolism, accompanied by enhanced IL-1 signaling
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