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Early studies have confirmed that G6PD-deficient patients may display impeded neutrophilic function and impaired NETs, potentially affecting the immune systems ability to clear infections [15,16,17,18]
In response to nutrients, enteroendocrine L-cells and K-cells produce and release the incretin hormones including glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP), respectively [2]
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